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Mutation in BT2391 provides a competitive advantage over the wild-type (WT) in the gut by enhancing mucin glycan utilization and adhesion to mucosal epithelial cells.
Lee et al. 2022 Journal of Microbiology 60(2) 215-223
Colonization of the BT2391 mutant increased susceptibility to colitis in low-fiber diet-fed mice, suggesting that BT2391 may be involved in maintaining a balance between mucosal colonization and gut homeostasis.
Lee et al. 2022 Journal of Microbiology 60(2) 215-223
Deletion of [BT2391](brain://Oqz0b9Qo…