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MK-0616 produced dose-dependent reductions in LDL-cholesterol, non-HDL-cholesterol, and apolipoprotein (apo) B levels. Furthermore, MK-0616 modestly lowered lipoprotein (a) [Lp(a)].
Safety and tolerability studies reported to date are promising. MK-0616 may offer advantages over injectable anti-PCSK9 therapies in terms of ease of dosing, patient preference and cost. The results from phase III trials of MK-0616 on cardiovascular outcomes are awaited with interest.
see also:
[Proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibitors & Hypercholesterolemia (HChol)](brain://kQ2OFILxQ0qQrl9se9HExQ/ProproteinConvertaseSubtilisinkexinType9Pcsk9Inhibitors…