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The inflammation in T2D patients originates from T helper 17 (Th17) cells, likely due to defects in the fatty acid metabolic pathways that trigger Th17 inflammation in T2D.
Treg and Th17 cells are imbalanced during inflammatory and pathological conditions such as obesity.
Some studies showed that peripheral Treg and Th17 cells already exhibit increased imbalance with worsening glucose metabolism disorder in children with obesity.
Microbiota of Synbiotic Treated Mice Increases the Number of Intestinal RORgt-Th17 Cells and Prevents Glucose Intolerance
see also:
Type 2 Diabetes (T2D)…