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EGFR was demonstrated to be a substrate of ST6Gal 1 Sialyltransferase (ST6Gal-I), and the sialylated EGFR had an impact on the PI3K/Akt pathway
ST6Gal 1 Sialyltransferase (ST6Gal-I) axis is involved in the progression of T-ALL/MDR
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