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Direct effects of Lipopolysaccharides (LPS) on Pancreatic Ductal Adenocarcinoma (PDAC) cells have been observed, where Lipopolysaccharides (LPS) can accelerate tumor progression and invasion, decrease tumor suppressor activity, and increase migratory ability. Lipopolysaccharides (LPS) exposure led to differential gene expression in PDAC cells, affecting pathways such as PI3K/Akt signaling, which is a known oncogenic driver
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[Carcinogenesis / Oncogenesis / Tumorigenesis & Lipopolysaccharides (LPS)](brain://miKKMPLjtUiw-8S1Sxqwlw/CarcinogenesisOncogenesisTumorigenesisLipopolysac…