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Viruses are recognized by receptors of the innate immune system, which leads, among other things, to the induction of antiviral type I interferons (IFNs) (1). After their release, type I IFNs recruit professional immune cells and induce a broad transcriptional program of hundreds of interferon-stimulated genes (ISGs) in an autocrine and paracrine manner.
Interferon-stimulated genes (ISGs) not only encode antiviral restriction factors but also nucleic acid receptors and …