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Treatment of mice, where colitis was induced by DNBS, with Lactococcus lactis LL-ProBi-H1:hCAP18 reduced the severity of colitis symptoms. The protective effects were accompanied by increased levels of IL17A and IL10 in mesenteric lymph node cells. L. lactis administration altered the abundance of Lachnospiraceae and Muribaculaceae. However, fecal transplant from L. lactis‑treated mice did not improve DNBS‑induced symptoms in recipient mice.
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