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Tislelizumab was engineered to minimize binding to FcγR on macrophages to abrogate antibody-dependent phagocytosis, a mechanism of T-cell clearance and potential resistance to anti-PD-1 therapy.
Liu & Wu 2020 Expert Opin. Investig. Drugs 29: 1355-1364
Tislelizumab has a higher affinity to CD279 (Programmed cell death protein 1 (PD-1) than pembrolizumab and nivolumab, potentially due to its differential PD-1 binding orientation.
Liu & Wu 2020 Expert Opin. Investig. Drugs 29: 1355-1364
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